Review



anti ercc8  (Proteintech)


Bioz Verified Symbol Proteintech is a verified supplier  
  • Logo
  • About
  • News
  • Press Release
  • Team
  • Advisors
  • Partners
  • Contact
  • Bioz Stars
  • Bioz vStars
    • 93
      Buy from Supplier

    Structured Review

    Proteintech anti ercc8
    Anti Ercc8, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 15 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+ercc8/ERCC2+Antibody/pmc12806303-228-35-36
    Average 93 stars, based on 15 article reviews
    anti ercc8 - by Bioz Stars, 2026-09
    93/100 stars

    Images

    Related Articles

    other:

    Article Title: Arid1a Deficiency Drives Aristolochic Acid-Induced Liver Tumorigenesis through Ctnnb1 Mutation and Defective Nucleotide Excision Repair.
    Article Snippet: Antibodies and Reagents: The antibodies used were as follows: antiArid1a (Cell Signaling Technology, #12354; Abcam, ab182560), antiCk19 (Abcam, ab52625), anti-GS (Abcam, ab73593), anti-Ki67 (Cell Signaling Technology, #12202), anti-β-Catenin (Cell Signaling Technology, #9582), anti-Xpc (Abclonal, A8354), anti-Xpa (Proteintech, 16462-1-AP), anti-Ercc8 (Proteintech, 10818-1-AP), anti-Brg1 (Abcam, ab110641), antiNqo1 (Abmart, T56710F), anti-Nrf2 (Cell Signaling Technology, #12721), anti-HDAC1 (Abcam, ab280198), anti-histone H4 (acetyl Lys8) (Abcam, ab15823), anti-acetyl-histone H4 (Millipore, 06–598), anti-RNA polymerase II (Millipore, 05–623) and anti-Gapdh (Proteintech, 60004-1-Ig).

    Article Title: Arid1a Deficiency Drives Aristolochic Acid‐Induced Liver Tumorigenesis through Ctnnb1 Mutation and Defective Nucleotide Excision Repair
    Article Snippet: The antibodies used were as follows: anti‐Arid1a (Cell Signaling Technology, #12354; Abcam, ab182560), anti‐Ck19 (Abcam, ab52625), anti‐GS (Abcam, ab73593), anti‐Ki67 (Cell Signaling Technology, #12202), anti‐β‐Catenin (Cell Signaling Technology, #9582), anti‐Xpc (Abclonal, A8354), anti‐Xpa (Proteintech, 16462‐1‐AP), anti‐Ercc8 (Proteintech, 10818‐1‐AP), anti‐Brg1 (Abcam, ab110641), anti‐Nqo1 (Abmart, T56710F), anti‐Nrf2 (Cell Signaling Technology, #12721), anti‐HDAC1 (Abcam, ab280198), anti‐histone H4 (acetyl Lys8) (Abcam, ab15823), anti‐acetyl‐histone H4 (Millipore, 06–598), anti‐RNA polymerase II (Millipore, 05–623) and anti‐Gapdh (Proteintech, 60004‐1‐Ig).



    Similar Products

    anti ercc8  (Proteintech)
    93
    Proteintech anti ercc8
    Anti Ercc8, supplied by Proteintech, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+ercc8/ERCC2+Antibody/pmc12806303-228-35-36
    Average 93 stars, based on 1 article reviews
    anti ercc8 - by Bioz Stars, 2026-09
    93/100 stars
    		  Buy from Supplier
    rabbit anti-ercc8 [n2c2] internal ab  (GeneTex)
    90
    GeneTex rabbit anti-ercc8 [n2c2] internal ab
    Rabbit Anti Ercc8 [N2c2] Internal Ab, supplied by GeneTex, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+ercc8/antibody+of+erb++n2c2+/pm37216802-40-11-16
    Average 90 stars, based on 1 article reviews
    rabbit anti-ercc8 [n2c2] internal ab - by Bioz Stars, 2026-09
    90/100 stars
    		  Buy from Supplier
    ercc8  (Santa Cruz Biotechnology)
    93
    Santa Cruz Biotechnology ercc8
    Pedigree and genetic mutation of the family with keratoconus and congenital cataracts. ( A ) Pedigree. ( B ) Sanger sequencing confirms the c.394-398del (p. L132Nfs*6) mutation identified by WES (I-2 and II-1) and HRM (KC5). ( C ) Cross-species comparison of the region of <t>ERCC8</t> indicates that the identified mutation affects highly conserved residues. ( D ) Domain analysis of the p. L132Nfs*6 mutation. ( E ) The 3D structures of mutant and wild type protein.
    Ercc8, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+ercc8/CSA+Antibody/pmc09728497-57-9-11
    Average 93 stars, based on 1 article reviews
    ercc8 - by Bioz Stars, 2026-09
    93/100 stars
    		  Buy from Supplier
    rabbit anti ercc8  (Danaher Inc)
    86
    Danaher Inc rabbit anti ercc8
    Pedigree and genetic mutation of the family with keratoconus and congenital cataracts. ( A ) Pedigree. ( B ) Sanger sequencing confirms the c.394-398del (p. L132Nfs*6) mutation identified by WES (I-2 and II-1) and HRM (KC5). ( C ) Cross-species comparison of the region of <t>ERCC8</t> indicates that the identified mutation affects highly conserved residues. ( D ) Domain analysis of the p. L132Nfs*6 mutation. ( E ) The 3D structures of mutant and wild type protein.
    Rabbit Anti Ercc8, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+ercc8/pm36231052-91-46-48
    Average 86 stars, based on 1 article reviews
    rabbit anti ercc8 - by Bioz Stars, 2026-09
    86/100 stars
    		  Buy from Supplier
    ercc8  (Danaher Inc)
    86
    Danaher Inc ercc8
    Pedigree and genetic mutation of the family with keratoconus and congenital cataracts. ( A ) Pedigree. ( B ) Sanger sequencing confirms the c.394-398del (p. L132Nfs*6) mutation identified by WES (I-2 and II-1) and HRM (KC5). ( C ) Cross-species comparison of the region of <t>ERCC8</t> indicates that the identified mutation affects highly conserved residues. ( D ) Domain analysis of the p. L132Nfs*6 mutation. ( E ) The 3D structures of mutant and wild type protein.
    Ercc8, supplied by Danaher Inc, used in various techniques. Bioz Stars score: 86/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+ercc8/pm35063774-69-5-6
    Average 86 stars, based on 1 article reviews
    ercc8 - by Bioz Stars, 2026-09
    86/100 stars
    		  Buy from Supplier
    csa ercc8 mouse santa cruz  (Santa Cruz Biotechnology)
    93
    Santa Cruz Biotechnology csa ercc8 mouse santa cruz
    Pedigree and genetic mutation of the family with keratoconus and congenital cataracts. ( A ) Pedigree. ( B ) Sanger sequencing confirms the c.394-398del (p. L132Nfs*6) mutation identified by WES (I-2 and II-1) and HRM (KC5). ( C ) Cross-species comparison of the region of <t>ERCC8</t> indicates that the identified mutation affects highly conserved residues. ( D ) Domain analysis of the p. L132Nfs*6 mutation. ( E ) The 3D structures of mutant and wild type protein.
    Csa Ercc8 Mouse Santa Cruz, supplied by Santa Cruz Biotechnology, used in various techniques. Bioz Stars score: 93/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/anti+ercc8/CSA+Antibody/pmc07192910__41467_2020_15903_MOESM3_ESM-41-48-51
    Average 93 stars, based on 1 article reviews
    csa ercc8 mouse santa cruz - by Bioz Stars, 2026-09
    93/100 stars
    		  Buy from Supplier

    Image Search Results


    Pedigree and genetic mutation of the family with keratoconus and congenital cataracts. ( A ) Pedigree. ( B ) Sanger sequencing confirms the c.394-398del (p. L132Nfs*6) mutation identified by WES (I-2 and II-1) and HRM (KC5). ( C ) Cross-species comparison of the region of ERCC8 indicates that the identified mutation affects highly conserved residues. ( D ) Domain analysis of the p. L132Nfs*6 mutation. ( E ) The 3D structures of mutant and wild type protein.

    Journal: Investigative Ophthalmology & Visual Science

    Article Title: Insufficient Dose of ERCC8 Protein Caused by a Frameshift Mutation Is Associated With Keratoconus With Congenital Cataracts

    doi: 10.1167/iovs.63.13.1

    Figure Lengend Snippet: Pedigree and genetic mutation of the family with keratoconus and congenital cataracts. ( A ) Pedigree. ( B ) Sanger sequencing confirms the c.394-398del (p. L132Nfs*6) mutation identified by WES (I-2 and II-1) and HRM (KC5). ( C ) Cross-species comparison of the region of ERCC8 indicates that the identified mutation affects highly conserved residues. ( D ) Domain analysis of the p. L132Nfs*6 mutation. ( E ) The 3D structures of mutant and wild type protein.

    Article Snippet: Primary antibodies included those of collagen IV (ab6586; Abcam), ERCC8 (sc-376981; Santa Cruz), SOD1 (MABC684; Millipore), γ-H2AX (AP0099; Abclonal), NF-kB p65 (phosphor S536, ab76302; Abcam), MMP1 (ab52631; Abcam), Caspase 3 (D320074; Sangon), and GAPDH (KC-5G5; Kangchen, Shanghai, China).

    Techniques: Mutagenesis, Sequencing, Comparison

    Pathogenicity Assessment of ERCC8 Mutation

    Journal: Investigative Ophthalmology & Visual Science

    Article Title: Insufficient Dose of ERCC8 Protein Caused by a Frameshift Mutation Is Associated With Keratoconus With Congenital Cataracts

    doi: 10.1167/iovs.63.13.1

    Figure Lengend Snippet: Pathogenicity Assessment of ERCC8 Mutation

    Article Snippet: Primary antibodies included those of collagen IV (ab6586; Abcam), ERCC8 (sc-376981; Santa Cruz), SOD1 (MABC684; Millipore), γ-H2AX (AP0099; Abclonal), NF-kB p65 (phosphor S536, ab76302; Abcam), MMP1 (ab52631; Abcam), Caspase 3 (D320074; Sangon), and GAPDH (KC-5G5; Kangchen, Shanghai, China).

    Techniques: Mutagenesis, Sequencing, Control

    Expression of ERCC8 in different mouse tissues. ( A ) Representative figures of immunofluorescence staining of ERCC8 in the mouse eye (the upper was a panoramic image, and the lower were local images). ( B ) The mRNA expression levels of ERCC8 in different mouse tissues.

    Journal: Investigative Ophthalmology & Visual Science

    Article Title: Insufficient Dose of ERCC8 Protein Caused by a Frameshift Mutation Is Associated With Keratoconus With Congenital Cataracts

    doi: 10.1167/iovs.63.13.1

    Figure Lengend Snippet: Expression of ERCC8 in different mouse tissues. ( A ) Representative figures of immunofluorescence staining of ERCC8 in the mouse eye (the upper was a panoramic image, and the lower were local images). ( B ) The mRNA expression levels of ERCC8 in different mouse tissues.

    Article Snippet: Primary antibodies included those of collagen IV (ab6586; Abcam), ERCC8 (sc-376981; Santa Cruz), SOD1 (MABC684; Millipore), γ-H2AX (AP0099; Abclonal), NF-kB p65 (phosphor S536, ab76302; Abcam), MMP1 (ab52631; Abcam), Caspase 3 (D320074; Sangon), and GAPDH (KC-5G5; Kangchen, Shanghai, China).

    Techniques: Expressing, Immunofluorescence, Staining

    Mutant ERCC8 constructs and expression in human embryonic kidney 293 cells. ( A ) The fluorescence intensity of GFP after transfection for 48 hours. ( B ) mRNA expression levels of ERCC8 and TCR-related genes ( CSB , XPA , RPA , and XPG ) in overexpressed wild type and mutant cells. ( C ) Western blot results of ERCC8 protein in overexpressed wild type and mutant cells. ( D ) Statistical results of the Western blot; N = 3, *, P < 0.05.

    Journal: Investigative Ophthalmology & Visual Science

    Article Title: Insufficient Dose of ERCC8 Protein Caused by a Frameshift Mutation Is Associated With Keratoconus With Congenital Cataracts

    doi: 10.1167/iovs.63.13.1

    Figure Lengend Snippet: Mutant ERCC8 constructs and expression in human embryonic kidney 293 cells. ( A ) The fluorescence intensity of GFP after transfection for 48 hours. ( B ) mRNA expression levels of ERCC8 and TCR-related genes ( CSB , XPA , RPA , and XPG ) in overexpressed wild type and mutant cells. ( C ) Western blot results of ERCC8 protein in overexpressed wild type and mutant cells. ( D ) Statistical results of the Western blot; N = 3, *, P < 0.05.

    Article Snippet: Primary antibodies included those of collagen IV (ab6586; Abcam), ERCC8 (sc-376981; Santa Cruz), SOD1 (MABC684; Millipore), γ-H2AX (AP0099; Abclonal), NF-kB p65 (phosphor S536, ab76302; Abcam), MMP1 (ab52631; Abcam), Caspase 3 (D320074; Sangon), and GAPDH (KC-5G5; Kangchen, Shanghai, China).

    Techniques: Mutagenesis, Construct, Expressing, Fluorescence, Transfection, Western Blot

    Knockdown of ERCC8 in human corneal fibroblast cells. ( A ) The mRNA expression levels of ERCC8 after being transfected by ERCC8 siRNA. ( B ) The mRNA expression levels of TCR-related genes ( CSB , XPA , RPA , and XPG ) after being transfected by ERCC8 siRNA. ( C ) Western blot results of ERCC8, collagen Ⅳ, and SOD1 proteins after being transfected by ERCC8 siRNA. ( D ) Statistical results of the Western blot; N = 3, *, P < 0.05.

    Journal: Investigative Ophthalmology & Visual Science

    Article Title: Insufficient Dose of ERCC8 Protein Caused by a Frameshift Mutation Is Associated With Keratoconus With Congenital Cataracts

    doi: 10.1167/iovs.63.13.1

    Figure Lengend Snippet: Knockdown of ERCC8 in human corneal fibroblast cells. ( A ) The mRNA expression levels of ERCC8 after being transfected by ERCC8 siRNA. ( B ) The mRNA expression levels of TCR-related genes ( CSB , XPA , RPA , and XPG ) after being transfected by ERCC8 siRNA. ( C ) Western blot results of ERCC8, collagen Ⅳ, and SOD1 proteins after being transfected by ERCC8 siRNA. ( D ) Statistical results of the Western blot; N = 3, *, P < 0.05.

    Article Snippet: Primary antibodies included those of collagen IV (ab6586; Abcam), ERCC8 (sc-376981; Santa Cruz), SOD1 (MABC684; Millipore), γ-H2AX (AP0099; Abclonal), NF-kB p65 (phosphor S536, ab76302; Abcam), MMP1 (ab52631; Abcam), Caspase 3 (D320074; Sangon), and GAPDH (KC-5G5; Kangchen, Shanghai, China).

    Techniques: Knockdown, Expressing, Transfection, Western Blot

    Cell viability and DNA damage assays of HTK treated by H 2 O 2 after ERCC8 siRNA transfection. ( A ) Cell viability assay results. ( B ) Hoechst staining results. ( C ) Western blot results of ERCC8, γ- H2AX, and phospho p65 protein. ( D ) Statistical results of the Western blot; N = 3, *, P < 0.05.

    Journal: Investigative Ophthalmology & Visual Science

    Article Title: Insufficient Dose of ERCC8 Protein Caused by a Frameshift Mutation Is Associated With Keratoconus With Congenital Cataracts

    doi: 10.1167/iovs.63.13.1

    Figure Lengend Snippet: Cell viability and DNA damage assays of HTK treated by H 2 O 2 after ERCC8 siRNA transfection. ( A ) Cell viability assay results. ( B ) Hoechst staining results. ( C ) Western blot results of ERCC8, γ- H2AX, and phospho p65 protein. ( D ) Statistical results of the Western blot; N = 3, *, P < 0.05.

    Article Snippet: Primary antibodies included those of collagen IV (ab6586; Abcam), ERCC8 (sc-376981; Santa Cruz), SOD1 (MABC684; Millipore), γ-H2AX (AP0099; Abclonal), NF-kB p65 (phosphor S536, ab76302; Abcam), MMP1 (ab52631; Abcam), Caspase 3 (D320074; Sangon), and GAPDH (KC-5G5; Kangchen, Shanghai, China).

    Techniques: Transfection, Viability Assay, Staining, Western Blot

    Cell viability, apoptosis, and DNA damage assays of HLECs treated by H 2 O 2 after ERCC8 siRNA transfection. ( A ) Cell viability assay results. ( B ) The mRNA expression levels of TCR-related genes. ( C ) Representative figures of immunofluorescence staining of cell apoptosis. ( D ) Statistical results of the cell apoptosis analysis. ( E ) The mRNA expression levels of UPR-related genes. ( F ) Western blot results of SOD1, γ- H2AX, and caspase 3 protein. ( D ) Statistical results of the Western blot; N = 3, *, P < 0.05.

    Journal: Investigative Ophthalmology & Visual Science

    Article Title: Insufficient Dose of ERCC8 Protein Caused by a Frameshift Mutation Is Associated With Keratoconus With Congenital Cataracts

    doi: 10.1167/iovs.63.13.1

    Figure Lengend Snippet: Cell viability, apoptosis, and DNA damage assays of HLECs treated by H 2 O 2 after ERCC8 siRNA transfection. ( A ) Cell viability assay results. ( B ) The mRNA expression levels of TCR-related genes. ( C ) Representative figures of immunofluorescence staining of cell apoptosis. ( D ) Statistical results of the cell apoptosis analysis. ( E ) The mRNA expression levels of UPR-related genes. ( F ) Western blot results of SOD1, γ- H2AX, and caspase 3 protein. ( D ) Statistical results of the Western blot; N = 3, *, P < 0.05.

    Article Snippet: Primary antibodies included those of collagen IV (ab6586; Abcam), ERCC8 (sc-376981; Santa Cruz), SOD1 (MABC684; Millipore), γ-H2AX (AP0099; Abclonal), NF-kB p65 (phosphor S536, ab76302; Abcam), MMP1 (ab52631; Abcam), Caspase 3 (D320074; Sangon), and GAPDH (KC-5G5; Kangchen, Shanghai, China).

    Techniques: Transfection, Viability Assay, Expressing, Immunofluorescence, Staining, Western Blot